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Galactose protects against cell damage in mouse models of acute pancreatitis.
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Ca2+ release-activated Ca2+ channel blockade as a potential tool in antipancreatitis therapy.
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A novel role for Bcl-2 in regulation of cellular calcium extrusion.
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Mitochondrial function and malfunction in the pathophysiology of pancreatitis.
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Calmodulin protects against alcohol-induced pancreatic trypsinogen activation elicited via Ca2+ release through IP3 receptors.
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Inhibitors of Bcl-2 protein family deplete ER Ca2+ stores in pancreatic acinar cells.
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Two-photon permeabilization and calcium measurements in cellular organelles.
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Calcium elevation in mitochondria is the main Ca2+ requirement for mitochondrial permeability transition pore (mPTP) opening.
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Fatty acids, alcohol and fatty acid ethyl esters: Toxic Ca2+ signal generation and pancreatitis.
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Pancreatic protease activation by alcohol metabolite depends on Ca2+ release via acid store IP3 receptors.
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Calcium signalling and pancreatic cell death: apoptosis or necrosis?
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NAADP, cADPR and IP3 all release Ca2+ from the endoplasmic reticulum and an acidic store in the secretory granule area.
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New aspects of nuclear calcium signalling.
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NAADP mobilizes Ca2+ from a thapsigargin-sensitive store in the nuclear envelope by activating ryanodine receptors.
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The distribution of the endoplasmic reticulum in living pancreatic acinar cells.
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